Preliminary studies reveal conolidine could inhibit particular ion channels, decreasing neuronal excitability and restricting ache alerts. This system is especially appropriate in neuropathic ache, where abnormal signaling will cause persistent irritation. Also, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to soreness notion. Computational docking s... https://nielsonh602fhb6.vigilwiki.com/user